AtlasDevelopmentalDentinogenesis Imperfecta

Developmental Disorders

Dentinogenesis Imperfecta

aka DI · Hereditary Opalescent Dentin · Capdepont Teeth

Autosomal dominant disorder of dentin causing opalescent, amber-discoloured teeth prone to fracture.

Inheritance
Autosomal dominant
Gene
DSPP (Types II/III)
Radiograph
Obliterated pulp, short roots

§ overviewOverview

Heritable dentin defect due to DSPP gene mutation affecting both dentitions.

§ icdICD Classification

K00.5

§ etiologyEtiology

  • 01Mutation in DSPP gene (chromosome 4q21)
  • 02Autosomal dominant

§ epidemiologyEpidemiology

1:6,000 to 1:8,000 live births.

§ pathogenesisPathogenesis

Defective dentin sialophosphoprotein → disorganised collagen matrix and poor mineralisation → weak dentin.

§ clinicalClinical Features

  • 01Opalescent blue-grey or amber teeth
  • 02Bulbous crowns, cervical constriction
  • 03Rapid attrition with flat occlusal surfaces
  • 04Primary teeth more affected than permanent

§ differentialDifferential Diagnosis

  • 01Amelogenesis imperfecta
  • 02Dentin dysplasia
  • 03Tetracycline staining
  • 04Regional odontodysplasia

§ histopathHistopathology

  • 01Irregular, disorganised dentinal tubules
  • 02Abnormal predentin layer

§ radiographicRadiographic Features

  • 01Obliterated pulp chambers and root canals
  • 02Short, constricted roots
  • 03Periapical radiolucencies (asymptomatic)

§ investigationsInvestigations

  • 01Clinical + radiographic
  • 02Family history
  • 03Genetic testing

§ classificationClassification

  • 01Type I — associated with osteogenesis imperfecta
  • 02Type II — isolated (Shields type II)
  • 03Type III — Brandywine isolate (shell teeth)

§ treatmentTreatment

  • 01Stainless steel crowns (deciduous)
  • 02Full-coverage restorations
  • 03Overdentures
  • 04Implants after growth

§ complicationsComplications

  • 01Severe attrition
  • 02Spontaneous pulp exposure
  • 03Periapical pathology

§ prognosisPrognosis

Good with early intervention; lifelong restorative needs.

§ examKey Examination Points

  • 01Type I always with OI; Types II/III without OI
  • 02Pulp obliteration on radiograph is key
  • 03Enamel chips away due to poor DEJ support

§ revisionQuick Revision Summary

  • 01DSPP mutation
  • 02Bulbous crown, cervical constriction
  • 03Obliterated pulp on OPG

§ vivaBDS Viva Questions

  • 01What is the genetic basis of DI?
  • 02How does DI differ from DD?
  • 03Describe radiographic features of DI.

§ mcqsMCQs — Assessment (3)

Question 1

Which gene is mutated in isolated DI?

Question 2

DI Type I is associated with:

Question 3

Radiographic hallmark of DI:

References

  1. Shields ED. Birth Defects 1973;9:107
  2. Neville BW. Oral & Maxillofacial Pathology, 4e

Draft — pending faculty review. Educational use only; verify against current guidelines and primary sources before clinical application.