Oral Cancer
HPV-associated Oropharyngeal SCC
aka HPV-OPSCC
A biologically and epidemiologically distinct SCC of the tonsil and base of tongue driven by high-risk HPV-16; better prognosis than tobacco-related SCC and staged separately in AJCC 8.
§ overviewOverview
Squamous cell carcinoma of the oropharynx associated with transcriptionally active high-risk human papillomavirus (predominantly HPV-16).
§ icdICD Classification
C10
§ etiologyEtiology
- 01HPV-16 (>90% of HPV-OPSCC), HPV-18/33 less commonly
- 02Oral–oral and oral–genital sexual transmission
§ riskRisk Factors
- 01Number of lifetime sexual partners
- 02Oral sex
- 03Immunosuppression
- 04Male (M:F 4:1)
§ epidemiologyEpidemiology
Rising incidence in Western countries; now exceeds cervical cancer incidence in some populations; younger, non-smoking patients.
§ pathogenesisPathogenesis
HPV E6 inactivates p53; E7 inactivates pRB → cell cycle dysregulation; p16INK4a strongly upregulated as surrogate marker.
§ clinicalClinical Features
- 01Painless neck lump (cystic level II lymphadenopathy) often presenting feature
- 02Sore throat, dysphagia, referred otalgia
- 03Tonsillar asymmetry or base-of-tongue mass
§ differentialDifferential Diagnosis
- 01Tobacco-related HNSCC (p16 negative)
- 02Lymphoma
- 03Branchial cyst (in older adult with SCC — suspect metastasis)
§ investigationsInvestigations
- 01p16 IHC (surrogate; block staining >70% cells positive)
- 02HPV DNA PCR / RNA in situ hybridisation (confirmatory)
- 03MRI head-neck
- 04PET-CT for staging and unknown primary
- 05EUA with biopsy of tonsil / tongue base
§ ihcIHC / Special Stains
- 01p16 strongly positive (block-like)
- 02Confirm HPV with ISH/PCR to avoid false-positives
§ tnmTNM Staging
AJCC 8: separate staging for HPV-positive OPSCC — clinical N based on number and size of nodes; overall stage grouping generally lower than p16-negative disease.
§ treatmentTreatment
- 01Concurrent chemoradiation (cisplatin) — standard for locally advanced disease
- 02Transoral robotic surgery (TORS) with neck dissection for selected T1-T2 lesions ± adjuvant therapy
- 03De-escalation trials ongoing (reduced-dose RT)
§ complicationsComplications
- 01Mucositis, xerostomia, dysphagia, ORN, hypothyroidism
§ prognosisPrognosis
5-year overall survival ≈ 75–85% (vs 40–50% for p16-negative OPSCC).
§ preventionPrevention
- 01Prophylactic HPV vaccination (Gardasil-9)
- 02Safer sexual practices
§ examKey Examination Points
- 01Cystic neck node in adult non-smoker → work up for HPV-OPSCC
- 02Test p16 on every OPSCC
§ revisionQuick Revision Summary
- 01HPV-16 · p16+ · better prognosis · AJCC 8 separate staging · TORS or chemoRT
§ vivaBDS Viva Questions
- 01Why is p16 a surrogate for HPV?
- 02Staging difference in AJCC 8?
- 03Role of HPV vaccination in prevention?
§ mcqsMCQs — Assessment (3)
Question 1
Most common HPV type in OPSCC:
Question 2
IHC surrogate marker for HPV:
Question 3
Prognosis of HPV+ compared with HPV− OPSCC:
References
- Chaturvedi AK. J Clin Oncol 2011
- AJCC 8th Edition
Draft — pending faculty review. Educational use only; verify against current guidelines and primary sources before clinical application.