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OMFS-0128PathologyadvancedBuccal mucosa, palate, gingiva

Widespread painful oral erosions preceding skin blisters

47-year-old female

Draft — educational study aid, pending faculty review.

§Chief complaint

Painful sores throughout the mouth for 2 months, cannot eat

§Medical & dental history

  • 01No relevant drug history
  • 02Weight loss of 6 kg due to pain
  • 03Unable to brush due to pain; heavy plaque accumulation

§Clinical examination

  • 01Multiple ragged, irregular erosions on the buccal mucosa, soft palate and gingiva
  • 02Intact blisters rare; positive Nikolsky sign
  • 03Desquamative gingivitis
  • 04Two flaccid bullae on the scalp appearing last week

§ figuresFigures (1)

Fig. 1All about the skin تدعمه Google ترجمةترجمة Search DermNet NZ Search Home About Topics A–Z CME Donate Browse Contact Images Jobs Book Home » Topics A–Z » Drug-induced pemphigus Drug-induced pemphigus Author: Brian Wu, MD

Pemphigus new photo due to antihypertensive drug” · Masryyy · CC BY-SA 4.0 · Wikimedia Commons

Verified open-access images only, reproduced for education under their stated licences · no AI-generated or illustrative artwork · Pemphigus Vulgaris figures pending faculty review.

§Differential diagnosis

Pemphigus vulgarisPositive Nikolsky, suprabasal split, intercellular IgG
Mucous membrane pemphigoidSubepithelial split, tense blisters, ocular involvement
Erosive lichen planusStriae present, band-like infiltrate
Erythema multiformeAcute onset, target lesions, crusted lips

§Final diagnosis

Pemphigus vulgaris with mucosal-dominant presentation

Fragile blisters that rupture immediately, a positive Nikolsky sign, and a suprabasal split with acantholysis on histology, plus intercellular IgG in a fishnet pattern on direct immunofluorescence, separate pemphigus from the subepithelial pemphigoid group.

§Investigations

  • 01Perilesional incisional biopsy for H&E and a separate specimen in Michel medium for direct immunofluorescence
  • 02Indirect immunofluorescence and anti-desmoglein 1 and 3 ELISA
  • 03Baseline FBC, glucose, bone density before steroids

§Treatment plan

  • 01Systemic corticosteroids (prednisolone) as first-line
  • 02Steroid-sparing immunosuppression (azathioprine or mycophenolate); rituximab increasingly first-line
  • 03Topical high-potency steroid for oral lesions plus antifungal cover
  • 04Nutritional support and analgesia; dermatology co-management

§Histopathology report

Suprabasal intraepithelial split with acantholytic Tzanck cells and a row-of-tombstones basal layer. Direct immunofluorescence: intercellular IgG and C3 in a fishnet pattern.

§Follow-up & outcome

  • 01Oral lesions healed by 8 weeks on prednisolone and rituximab
  • 02Steroid tapered; on maintenance immunosuppression
  • 03Regular monitoring for steroid complications

§Clinical pearls / learning points

  • 01Oral lesions precede skin lesions in most cases of pemphigus vulgaris — dentists often diagnose it first.
  • 02Always take a second perilesional biopsy for direct immunofluorescence; H&E alone is not enough.
  • 03Untreated pemphigus vulgaris was historically fatal; prompt referral is essential.

§References

  • Murrell DF et al. Diagnosis and management of pemphigus: consensus statement.

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